a*******i
发帖数: 27 | 1 【 以下文字转载自 Biology 讨论区 】
发信人: SWJSJ (BC), 信区: Biology
标 题: 单开一贴，浙大范衡宇涉嫌更加严重的剽窃和学术不端！
发信站: BBS 未名空间站 (Thu Sep 17 23:38:04 2015, 美东)
靠，实在是看着不爽，凭啥清北撕逼就大家一致讨伐，浙大范衡宇更加恶劣的剽窃别人
成果却没人关注？下面转帖，是从张生家和谢灿那个帖子里看到的，其他几位ID不愿意
单开一贴，我实在看不下去，特开一贴。这个范衡宇据说还拿到了杰青，国内科研真是
尼玛无底线了啊。
如果有知道内情的，在此帖讨论吧，不然都被清北的刷屏了。当然，也许有其他隐情冤
枉了范衡宇，也可以内部人出来解释解释。如果确定是剽窃，这尼玛妥妥的应该上新语
丝头条啊。
以下转帖：
----------------------------------------------------------------------------
------------------------
发信人: forjoy (三分华盖), 信区: Biology
标 题: Re: 能拿炸药奖吗？
发信站: BBS 未名空间站 (Wed Sep 16 17:25:42 2015, 美东)
浙大的：
CORRECTIONS AND CLARIFICATIONS
Reports: “CRL4 complex regulates mammalian oocyte survival
and reprogramming by activation of TET proteins” by
C. Yu et al. (20 December 2013, p. 1518). The authors wish
to acknowledge contributions of Tadashi Nakagawa and Yue
Xiong (University of North Carolina—Chapel Hill). Nakagawa
and Xiong initiated the VprBP-Tet project and made substantial
contributions to the published study. Xiong’s lab generated
and provided us the VprBP conditional mutant mice on
the basis of direct collaboration in summer 2012. Nakagawa,
a postdoctoral fellow in Xiong’s lab, discovered the VprBPTet
interaction by immunoprecipitation and mass spectrometry
analysis of the VprBP immunocomplex in early 2012.
Nakagawa also discovered in 2012, through knocking down
and knockout VprBP in cultured human cells and in mouse
embryo fi broblasts, that the function of VprBP is critically
important for the activity of Tet. Xiong has communicated
these and other unpublished results to us in multiple communications
between August 2012 and April 2013. The HTML
version online has been corrected.
Letters to the Editor |
|
